Researchers at the University of Maryland and their collaborators have cloned a gene that makes wheat resistant to feeding by Hessian fly larvae and demonstrated its direct interaction with a protein produced by the insect. The research breaks through a longstanding barrier to understanding and engineering pest resistance in wheat. It is described in a research paper published Aug. 14, 2026, in the journal Science Advances.

When a wound does not heal properly, it leaves a scar. Similarly, mutations—which are permanent changes to genetic code—are often the result of damaged DNA that has not been properly repaired. Mutations can impede the function of genes and lead to disease and aging, but they are also the source of genetic variation, which allows new traits to emerge and facilitates the evolutionary process. Scientists still do not fully understand why some damaged DNA segments are successfully repaired while others are not.

When a wound does not heal properly, it leaves a scar. Similarly, mutations—which are permanent changes to genetic code—are often the result of damaged DNA that has not been properly repaired. Mutations can impede the function of genes and lead to disease and aging, but they are also the source of genetic variation, which allows new traits to emerge and facilitates the evolutionary process. Scientists still do not fully understand why some damaged DNA segments are successfully repaired while others are not.

An international research team led by plant scientist Professor Dr. Bart Thomma of the Institute of Plant Sciences at the University of Cologne, the MiBiNet Collaborative Research Center and the CEPLAS Cluster of Excellence for Plant Sciences has identified the region of the genome in the pathogenic fungus Verticillium dahliae responsible for producing a specific protein that the pathogen secretes during infection.

As parasites become increasingly resistant to existing drugs, researchers at the University of California, Riverside, are working to develop a new generation of treatments for two serious infectious diseases: malaria and babesiosis.

If you want to beat the heat of the summer sun, slowing down and doing less is a good strategy. However, researchers have long asked whether the same occurs at the cellular level. While cellular stress responses have been repeatedly studied, the impact of the environment on these responses remains relatively unknown.